OPCs play a crucial role in the repair and regeneration of the CNS. Following injury or disease, such as multiple sclerosis, OPCs can proliferate and migrate to the site of damage. There, they differentiate into oligodendrocytes, contributing to the remyelination of axons. This process is essential for restoring normal function and preventing further neurological deficits. Understanding the mechanisms that regulate OPC behavior and differentiation is a key area of research in histology and neurobiology.
